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Experiments were performed in SD rats to investigate the involvement of the CYP1B1 in the development of cholangiocarcinoma. The main enzyme target of the compound was evaluated through inhibition tests with carbon monoxide-reversed difference spectrum and comparison of the activity of various drugs including beta-naphthoflavone, benzo[a]pyrene, cyclophosphamide and p-nitroanisole (PNO). This compound exhibited a competitive substrate-deregulating effect on both hepatic and extrahepatic CYP1B1-induced activities in vitro. A very strong increase in CYP1B1-directed enzymatic activity was observed in the rats treated with beta-naphthoflavone or with beta-naphthoflavone plus carbon monoxide. This was accompanied by an increase in the rate of DNA synthesis measured by bromodeoxyuridine incorporation in the rat hepatic cells treated with beta-naphthoflavone. Moreover, the induction of proliferation of hepatic bile duct cells was induced in the rat liver by beta-naphthoflavone. The results of the present study may indicate the involvement of the CYP1B1 activation in the development of cholangiocarcinoma and the involvement of chemicals in the phenomenon.// Copyright 2020 the V8 project authors. All rights reserved.
// Use of this source code is governed by a BSD-style license that can be
// found in the LICENSE file.

// Flags: –force-new-debug-context –no-stress-opt

let g1 = newGlobal();
let g2 = newGlobal();
let g3 = newGlobal();
let g4 = newGlobal();

assertThrows(() => g1.x = g2, TypeError);
assertThrows(() => g1.x = g4, TypeError);
assertThrows(() => g1.y = g3, TypeError);
assertThrows(() => g1.
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